Aldosterone Nongenomically Produces NADPH Oxidase–Dependent Reactive Oxygen Species And Induces Myocyte Apoptosis
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Non-genomic Effects of Aldosterone - Nottingham
H (2008). Aldosterone nongenomically produces NADPH oxidase-dependent reactive oxygen species and induces myocyte apoptosis, Hypertens. Res. 31: 363-375. Ivanova L, Bernhardt R, Bernhardt I (2008). Nongenomic effect of aldosterone on ion transport pathways of red blood cells, Cell Physiol Biochem. 22: 269-78.
Aldosterone Nongenomically Produces NADPH Oxidase Dependent
aldosterone (10 7 mol/L) increased the myocyte apoptosis (2.3 fold, p<0.001), coincident with the activation of caspase-3 (1.4 fold, p <0.05), compared with the serum-deprived control after 48 h
Networking Between Systemic Angiotensin II and Cardiac
activation of gp91phox-containing NADPH oxidase, genera-tion of reactive oxygen species, activation of MMPs (MMP2 and MMP9), and increased collagen deposition, without overt inflammation. Interestingly these phenomena may not depend on increased cardiac aldosterone production, because trans-genic mice that overexpress the terminal enzyme of
myocyte monlayers. Aldosterone nongenomically produces NADPH oxidase dependent reactive oxygen species and induces myocyte apoptosis. Early cardiac involvement in senile systemic amyloidosis Dual SPECT imaging with Tc 99m pyrophosphate and Tl 201 of circumferential subendocardial myocardial infarction in aortic stenosis without